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TitleHepatic Encephalopathy in Chronic Liver Failure [electronic resource] / edited by Livio Capocaccia, Joseph E. Fischer, Filippo Rossi-Fanelli
ImprintBoston, MA : Springer US, 1984
Connect tohttp://dx.doi.org/10.1007/978-1-4684-4787-3
Descript XII, 400 p. online resource

SUMMARY

The meeting which took place in Rome on November 19th and 20th of 1982 is easily the best meeting on hepatic coma that I have ever attended, and I have attended many. It was an exceedingly we- planned meeting with prolonged opportunity for discussion, and there was genuine interplay and exchange of ideas (not the usual picture of a rushed meeting with investigators presenting their own point of view and talking past each other without a meaningful exยญ change) which took place in Rome. My co-editors and I hope that the published transcript, which of course can only reflect what transpired in Rome on those two days, does justice to a very intellectually exciting and gratifying exยญ change of ideas. L. Capocaccia, J. E. Fischer and F. Rossi-Fanelli v CONTENTS Introduction 1 SECTION 1: PATHOGENETIC PROBLEMS IN HEPATIC ENCEPHALOPATHY Ammonia: The old and the new โ{128}ข โ{128}ข . โ{128}ข โ{128}ข โ{128}ข . โ{128}ข . โ{128}ข โ{128}ข โ{128}ข . โ{128}ข . โ{128}ข 5 L. Zieve Role of synergism in the pathogenesis of hepatic encephalopathy โ{128}ข โ{128}ข โ{128}ข โ{128}ข . โ{128}ข โ{128}ข . . . . โ{128}ข 15 L. Zieve y-Aminobutyric acid receptors in experimental hepatic encephalopathy โ{128}ข. โ{128}ข. โ{128}ข โ{128}ข 25 M. L. Zeneroli, ̃. Baraldi,and E. Ventura A possible role for excitatory neurotoxic amino acids in the pathogenesis of hepatic enยญ 41 cephalopathy โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข โ{128}ข F. Moroni, G. Lombardi, G. Moneti, D. Pellegrini and C


CONTENT

Section 1: Pathogenetic Problems in Hepatic Encephalopathy -- Ammonia: The old and the new -- Role of synergism in the pathogenesis of hepatic encephalopathy -- ?-Aminobutyric acid receptors in experimental hepatic encephalopathy -- A possible role for excitatory neurotoxic amino acids in the pathogenesis of hepatic encephalopathy -- LThe development of the false neurotransmitter concept of hepatic encephalopathy -- Studies relating to a theoretical understanding of altered blood-brain barrier transport in liver disease -- Amino acid transport in isolated brain capillaries -- Cerebro-spinal fluid amino acid pattern in hepatic encephalopathy -- Alterations of plasma and brain tryptophan in hepatic encephalopathy: a study in humans and in experimental animals -- New methods for the determination of serum and breath mercaptans and its application in liver cirrhosis and hepatic encephalopathy -- Plasma influence on the altered blood-brain barrier to amino acids after portacaval shunt: Preliminary results -- Hepatic encephalopathy is associated with decreased numbers of receptors for excitatory aminoacid neurotransmitters -- Brain gamma amino-butyric acid in acute hepatic encephalopathy in dogs following hepatectomy with or without abdominal evisceration -- Section 2: Metabolic and Prophylactic Aspects of Hepatic Encephalopathy -- Carbohydrate metabolism in liver disease -- Proteins and amino acids in liver failure -- The metabolic fate of branched chain amino acids -- Long term dietary supplement with branched chain amino acids: a new approach in the prevention of hepatic encephalopathy: Results of a controlled study in cirrhotics with portocavai anastomosis -- Muscle biopsy studies on malnutrition in patients with liver cirrhosis: Preliminary results of long-term treatment with a branched-chain amino acid enriched diet -- The role of muscle protein catabolism in the plasma amino acid profile of cirrhotics -- Plasma prolactin in liver cirrhosis -- Short-term branched chain amino acid parenteral nutrition in liver cirrhosis: A dose-related effect on plasma amino acids and nitrogen balance -- Nutritional effects of branched-chain ketoanalogues in chronic hepatic and renal failure: A preliminary report -- Section 3: Assessment and Evaluation of Hepatic Encephalopathy -- Summary of the contributions -- Visual evoked potentials in the quantitative assessment of portal-systemic encephalopathy and its preclinical stage -- Section 4: Therapeutic Aspects of Hepatic Encephalopathy -- Bromocriptine in the treatment of chronic hepatic encephalopathy -- Bromocriptine to treat portal systemic encephalopathy -- Lactitol, a potential second generation disaccharide drug for the treatment of portal-systemic encephalopathy -- Lactose to treat acute and chronic portal systemic encephalopathy -- Do branched-chain amino acids have a role in the treatment of hepatic encephalopathy? -- Treatment of hepatic encephalopathy by infusion of a modified amino acid solution: Results of a controlled study in 47 cirrhotic patients -- Efficacy of branched-chain amino acids in the treatment of hepatic encephalopathy -- Branched chain amino acid enriched solutions in the treatment of hepatic encephalopathy: A controlled trial -- Branched-chain amino acids in the treatment of severe hepatic encephalopathy -- Prophylaxis of hepatic encephalopathy after portacaval anastomosis using branched chain amino acid mixtures -- Effective treatment of latent porto-systemic encephalopathy with oral branched chain amino acids -- Effect of vegan and meat protein diets in mild chronic portal-systemic encephalopathy -- 14C] -L-Valine binding to post mortem frontal cortex homogenates in hepatic encephalopathy -- Regional analysis of norepinephrine, dopamine and amino acids in the brain 18 hours after hepatectomy and infusion of branched-chain amino acids -- Comment to oral BCAA trials -- Contributors


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